It has been demonstrated from work by Shinya Yamanaka (stem cell researcher and a Nobel Prize laureate) and many other scientists, including Dr. Sebastiano at Stanford, who work in the field, that stem cells have the ability to heal as well as rejuvenate and erase damage from a myriad of environmental and genetic conditions.1,2,3

With the ongoing advancement of stem cell medical treatments and discoveries, we can now envision the reversing of certain diseases and conditions for patients of all ages. This promise of rejuvenation, the relief of pain and inflammation, has become a scientific reality and creates the possibility to address certain human diseases in a completely different way, one without pills. One where we utilize the power and utility of our cells.

Personalized Stem Cells (PSC) and scientists around the world are working to create novel approaches to medicine based on the emerging field of stem cell medicine. Over the last decade, scientists and clinicians have begun to quantify these cellular benefits and are now understanding the mechanism behind stem cells’ medicinal pathways that have the ability to regenerate and rejuvenate our general well-being.

PSC will continue to share and build on the remarkable development of stem cells as we learn more on how they regulate the numerous cell healing processes in the human body. Our goals will always be focused on uses of stem cells for the repair and in some cases the reversal of the damage that comes from life and aging. Cells are the foundation of our wellbeing, and this field provides an opportunity for new medical treatments that allow patients to improve their quality of life naturally.


  1. Takahashi K, Yamanaka S. Induction of pluripotent stem cells from mouse embryonic and adult fibroblast cultures by defined factors. Cell. 2006 Aug 25;126(4):663-76. doi: 10.1016/j.cell.2006.07.024. Epub 2006 Aug 10. PMID: 16904174.
  3. Hung, G., Ashvetiya, T., Leszczynska, A. et al. Paracrine-mediated rejuvenation of aged mesenchymal stem cells is associated with downregulation of the autophagy-lysosomal pathway. npj Aging 8, 10 (2022).